Amyloid β-Protein (35-25)

Description:

The reverse sequence of Aβ 25-35 is used as inactive control.

Sequence:

MLGIIAGKNSG

General

References

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Name	Amyloid β-Protein (35-25)
Category	Beta-Amyloidand Related Peptides
One Letter Code	MLGIIAGKNSG
Three Letter Code	{Met}{Leu}{Gly}{Ile}{Ile}{Ala}{Gly}{Lys}{Asn}{Ser}{Gly}
Molecular Weight	1060.280
Application	Alzheimer's Disease

Eufemi, Margherita, et al. "Acetylation and phosphorylation of STAT3 are involved in the responsiveness of microglia to beta amyloid." Neurochemistry international 81 (2015): 48-56.

Uzakov, Shukhrat S., et al. "Lentiviral-mediated overexpression of nerve growth factor (NGF) prevents beta-amyloid [25–35]-induced long term potentiation (LTP) decline in the rat hippocampus." Brain research 1624 (2015): 398-404.

Rice, Ann C., et al. "Mitochondrial DNA copy numbers in pyramidal neurons are decreased and mitochondrial biogenesis transcriptome signaling is disrupted in Alzheimer's disease hippocampi." Journal of Alzheimer's Disease 40.2 (2014): 319-330.

Deleglise, Bérangère, et al. "β-amyloid induces a dying-back process and remote trans-synaptic alterations in a microfluidic-based reconstructed neuronal network." Acta neuropathologica communications 2.1 (2014): 145.

Dal Pra, Ilaria, et al. "The Aβ peptides-activated calcium-sensing receptor stimulates the production and secretion of vascular endothelial growth factor-A by normoxic adult human cortical astrocytes." Neuromolecular medicine 16.4 (2014): 645-657.

Kim, Jin Hee. "Brain‑derived neurotrophic factor exerts neuroprotective actions against amyloid β‑induced apoptosis in neuroblastoma cells." Experimental and therapeutic medicine 8.6 (2014): 1891-1895.