Beta - Amyloid (25 - 35)

Description:

Aβ (25-35) is the main factor responsible for Aβ neurotrophic and neurotoxic effects. The sequence GSNKGAIIGLM is homologous to a region in peptides of the tachykinin family.

Sequence:

GSNKGAIIGLM

General

References

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Name	Beta - Amyloid (25 - 35)
Category	Beta-Amyloidand Related Peptides
One Letter Code	GSNKGAIIGLM
Three Letter Code	{Gly}{Ser}{Asn}{Lys}{Gly}{Ala}{Ile}{Ile}{Gly}{Leu}{Met}
Molecular Weight	1060.300
Application	Alzheimer's Disease

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Accardo, Angelo, et al. "Superhydrophobic surfaces boost fibril self-assembly of amyloid β peptides." ACS applied materials & interfaces 7.37 (2015): 20875-20884.

Jerónimo-Santos, André, et al. "Brain-derived neurotrophic factor mediates neuroprotection against A β-induced toxicity through a mechanism independent on adenosine 2A receptor activation." Growth Factors 33.4 (2015): 298-308.

Kwon, Jaeyoung, et al. "Spiroindole alkaloids and spiroditerpenoids from Aspergillus duricaulis and their potential neuroprotective effects." Journal of natural products 78.11 (2015): 2572-2579.

Li, Li, et al. "The neuroprotective effects of decursin isolated from Angelica gigas nakai against amyloid β-protein-induced apoptosis in PC 12 cells via a mitochondria-related caspase pathway." Neurochemical research 40.8 (2015): 1555-1562.

Eufemi, Margherita, et al. "Acetylation and phosphorylation of STAT3 are involved in the responsiveness of microglia to beta amyloid." Neurochemistry international 81 (2015): 48-56.

Uzakov, Shukhrat S., et al. "Lentiviral-mediated overexpression of nerve growth factor (NGF) prevents beta-amyloid [25–35]-induced long term potentiation (LTP) decline in the rat hippocampus." Brain research 1624 (2015): 398-404.

Correani, Virginia, et al. "Reversible redox modifications in the microglial proteome challenged by beta amyloid." Molecular BioSystems 11.6 (2015): 1584-1593.